Daytime napping and mortality risk. When sleep may be a sign of an underlying health problem
A new study suggests that longer and more frequent naps in older adults may be a marker of higher mortality risk, especially when they occur in the morning.
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A nap does not always mean recovery
Daytime napping is common among older adults and, at first glance, may look like a simple response to fatigue. A short nap can improve alertness, reduce sleepiness, and create a subjective feeling of restored energy. The problem begins when naps become long, frequent, or occur at an unusual time of day.
A new study published in JAMA Network Open suggests that napping patterns may be more than just a habit. In older adults, longer and more frequent daytime naps were associated with a higher risk of all-cause mortality. The timing of naps was especially interesting: morning naps were linked to a higher risk than afternoon naps.
This does not mean that napping itself “shortens life.” A more likely interpretation is that excessive daytime sleepiness may be a marker of underlying health processes: sleep disorders, circadian disruption, chronic inflammation, cardiovascular disease, or neurodegeneration.
Study details
- Publication title: Objectively Measured Daytime Napping Patterns and All-Cause Mortality in Older Adults.
- Authors: Chenlu Gao, Ruifeng Cai, Xiaoyan Zheng, Arnav Gaba, Lei Yu, Aron S. Buchman, David A. Bennett, Lei Gao, Kun Hu, Peng Li.
- Publication year: 2026.
- Journal: JAMA Network Open.
- Identifiers: DOI was not provided in the source material.
- Publication link: JAMA Network Open.
- Study type and design: observational study using objective activity and sleep measurement through wrist actigraphy.
- Population and sample: 1,338 participants aged 56 years or older, recruited from retirement communities, senior and subsidized housing, and church groups in northern Illinois.
- Data source: Rush Memory and Aging Project, initiated in 1997, with mortality follow-up through 2025.
- Exposure measurement: daytime napping patterns assessed using wrist-worn device data recorded continuously for up to 14 days.
- Primary outcome: association between nap duration, frequency, timing, and variability and all-cause mortality risk.
- Funding and conflicts of interest: not described in the provided material.
This context matters because the study did not rely only on participants’ self-reported sleep habits. The authors used device-based data that made it possible to capture real daytime rest patterns, although actigraphy is still not a perfect tool for distinguishing sleep from very quiet wakefulness.
Duration, frequency, and timing of naps mattered
The analysis showed that mortality risk was mainly associated with nap duration and nap frequency. Variability in nap duration from day to day, however, was not significantly associated with mortality risk.
The authors translated the strength of these associations into age-equivalent comparisons to make the scale easier to understand. This does not mean that a nap biologically “adds years,” but that statistically it was linked to a similar increase in risk as a certain difference in age.
The key observations were as follows:
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Each additional hour of daytime napping corresponded to a risk similar to being approximately 1.1 years older. This suggests that very long naps may be an important signal of impaired sleep regulation or poorer overall health.
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Each additional daily nap corresponded to a risk similar to being approximately 0.6 years older. Frequent daytime sleep episodes may therefore not be a neutral habit, especially if they occur despite sufficient nighttime sleep.
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Morning naps, between 9 AM and 1 PM, were associated with higher risk than naps taken in the early afternoon. The authors compared this difference to a risk equivalent to being approximately 2.5 years older.
The timing of a nap may be important for interpretation. An afternoon drop in energy is relatively natural and, in some cultures, built into the rhythm of the day. A morning nap, however, may suggest that nighttime sleep is not fulfilling its restorative role or that the body is operating in a rhythm disrupted by disease, inflammation, or circadian misalignment.
What may connect napping with health risk
The simplest explanation would be that older adults nap more because they are more tired. But that is only the beginning of the question. The more important issue is: why does the body need more and more sleep during the day?
The study authors and previous literature point to several possible mechanisms. Each of them shows that a nap may be less of a direct cause of risk and more of a visible symptom.
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Nighttime sleep problems may lead to daytime sleepiness. People with poorer sleep quality, frequent awakenings, or breathing-related sleep problems may compensate during the day. Even though the statistical models accounted for nighttime sleep duration and quality, some disorders may still remain undetected.
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Circadian disruption may shift sleepiness to the wrong time of day. Morning naps may suggest that the body is not maintaining a stable sleep-wake rhythm. During aging, circadian rhythms often weaken, and their disruption may affect metabolism, blood pressure, and recovery.
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Chronic disease may cause fatigue that leads to napping. Subclinical or undiagnosed conditions may increase sleepiness before they are formally recognized. In this scenario, napping is a warning sign rather than the direct source of risk.
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The cardiovascular system may be part of this connection. Long daytime naps have previously been linked to cardiovascular risk factors and cardiovascular disease. Sleep disruption and circadian misalignment may contribute to higher blood pressure, a pro-inflammatory state, and atherosclerotic processes.
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Chronic inflammation may increase fatigue. Earlier observational studies linked daytime napping with higher levels of inflammatory markers such as CRP. Inflammation may act like a biological “brake,” lowering energy and increasing the need for sleep during the day.
In practice, this means that a sudden increase in the need for naps in an older person should not automatically be treated as a normal part of aging. It may be a reason to look more closely at nighttime sleep quality, circadian rhythm, activity level, inflammation, and chronic disease.
This is correlation, not proof of causation
The most important limitation of the study is simple: it was observational. Based on these data, it is not possible to conclude that naps themselves cause a higher risk of death. The authors clearly emphasize that this is correlation, not proof of a cause-and-effect relationship.
This distinction matters. A short, occasional nap after a poor night of sleep or intense exertion is not the same as daily, prolonged sleepiness in an older adult. The study suggests instead that napping patterns may help identify people whose health should be assessed more carefully.
The most practical conclusions are cautious:
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Not every nap is a problem. Short naps, especially in the afternoon, may be neutral or even helpful for some people, particularly when they do not disrupt nighttime sleep.
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Long and frequent naps should be treated as a signal worth monitoring. If an older person increasingly falls asleep during the day, it may point to impaired recovery, chronic disease, or undetected sleep disorders.
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Morning naps may deserve particular attention. Sleepiness early in the day may suggest circadian disruption or insufficiently restorative nighttime sleep.
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Wearable devices may help detect changes. Watches and activity trackers do not replace medical diagnostics, but they can reveal trends that used to be difficult to capture.
The most reasonable interpretation is therefore not that naps are the enemy. Rather, in older adults, their duration, frequency, and timing may be an important biomarker of lifestyle and health status, especially when they change suddenly or begin to dominate the rhythm of the day.
Sources
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Gao, C., Cai, R., Zheng, X., Gaba, A., Yu, L., Buchman, A. S., Bennett, D. A., Gao, L., Hu, K., & Li, P. (2026). Objectively Measured Daytime Napping Patterns and All-Cause Mortality in Older Adults. JAMA Network Open, 9(4), e267938.
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Zhang, Z., Xiao, X., Ma, W., & Li, J. (2020). Napping in Older Adults: A Review of Current Literature. Current Sleep Medicine Reports, 6(3), 129–135.
-
Sun, J., Ma, C., Zhao, M., Magnussen, C. G., & Xi, B. (2022). Daytime napping and cardiovascular risk factors, cardiovascular disease, and mortality: A systematic review. Sleep Medicine Reviews, 65, 101682.
-
Li, P., Gao, L., Yu, L., Zheng, X., Ulsa, M. C., Yang, H. W., Gaba, A., Yaffe, K., Bennett, D. A., Buchman, A. S., Hu, K., & Leng, Y. (2023). Daytime napping and Alzheimer’s dementia: A potential bidirectional relationship. Alzheimer’s & Dementia, 19(1), 158–168.
-
Leng, Y., Wainwright, N. W., Cappuccio, F. P., Surtees, P. G., Hayat, S., Luben, R., Brayne, C., & Khaw, K. T. (2014). Daytime napping and the risk of all-cause and cause-specific mortality: a 13-year follow-up of a British population. American Journal of Epidemiology, 179(9), 1115–1124.
-
Milner, C. E., & Cote, K. A. (2009). Benefits of napping in healthy adults: impact of nap length, time of day, age, and experience with napping. Journal of Sleep Research, 18(2), 272–281.
-
Bonnet, M. H. (1986). Performance and sleepiness as a function of frequency and placement of sleep disruption. Psychophysiology, 23(3), 263–271.
-
Wang, M., Xiang, X., Zhao, Z., Liu, Y., Cao, Y., Guo, W., Hou, L., & Jiang, Q. (2024). Association between self-reported napping and risk of cardiovascular disease and all-cause mortality: A meta-analysis of cohort studies. PLOS ONE, 19(10), e0311266.
-
Covassin, N., Bukartyk, J., Singh, P., Calvin, A. D., St Louis, E. K., & Somers, V. K. (2021). Effects of Experimental Sleep Restriction on Ambulatory and Sleep Blood Pressure in Healthy Young Adults: A Randomized Crossover Study. Hypertension, 78(3), 859–870.
-
Leng, Y., Ahmadi-Abhari, S., Wainwright, N. W., Cappuccio, F. P., Surtees, P. G., Luben, R., Brayne, C., & Khaw, K. T. (2014). Daytime napping, sleep duration and serum C reactive protein: a population-based cohort study. BMJ Open, 4(11), e006071.
-
Wright, F., Hammer, M., Paul, S. M., Aouizerat, B. E., Kober, K. M., Conley, Y. P., Cooper, B. A., Dunn, L. B., Levine, J. D., D’Eramo Melkus, G., & Miaskowski, C. (2017). Inflammatory pathway genes associated with inter-individual variability in the trajectories of morning and evening fatigue in patients receiving chemotherapy. Cytokine, 91, 187–210.